The rabbits with what appear to be “tentacles,” “horns,” or black spines growing from their heads and mouths are exhibiting symptoms of a viral infection—specifically, the Shope papilloma virus (SPV), also known as cottontail rabbit papillomavirus (CRPV).
There’s no direct literature linking SPV to sodium or SCN⁻ deficiency, but terrain logic suggests:
Sodium is essential for fluid balance, nerve signaling, and immune modulation. Deficiency can impair epithelial integrity and immune resilience—making viral colonization easier.
SCN⁻, a product of dietary cyanogenic compounds and sulfur metabolism, is a known antiviral, antibacterial, and anti-inflammatory molecule. It’s part of the lactoperoxidase system in saliva and mucosa—exactly where SPV lesions appear.
If wild rabbits are exposed to soil depletion, synthetic feed runoff, or disrupted plant biochemistry, their access to sodium and SCN⁻ precursors could be compromised.
🌿 What would wild rabbits need to restore sodium and SCN⁻ levels?
🧂 Sodium Sources
- Mineral-rich plants: Dandelion greens, clover, alfalfa
- Soil ingestion: Rabbits naturally ingest trace minerals via root nibbling and grooming
- Salt licks: In mineral-rich zones, wild rabbits may seek natural salt deposits
🧪 SCN⁻ Precursors
- Glucosinolates from wild brassicas (mustard greens, wild cabbage)
- Cyanogenic glycosides from clover, sorghum, cassava leaves (in moderation)
- Sulfur-rich plants: Garlic mustard, wild onions, and certain legumes
These compounds are metabolized into SCN⁻ via enzymatic pathways involving rhodanese and sulfur transferases—enzymes that require adequate sulfur, selenium, and B vitamins.
Leave a Reply